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So far, exposures to these chemistries have been associated with cancers, but the causes of the compound are still not known. Recent research conducted by Professor Ashok Venkitaraman, Director of the Medical Research Council Cancer Unit at the University of Cambridge, has used GM HCTC and cells from patient with a defective copy of the chest carcinogen bacterium BCRCA2 to find the mechanisms by which increased aldehyde exposures to carcinoma could occur.
Damages to our genetic material, often caused by the division of our body's own cell lines, can cause cancer, but our own immune system is able to help repair the damages. Professor Venkitaraman and fellow researchers, however, found that Aldehyd exposition also resolves these defense pathways in normally functioning intact cell lines, but humans who have acquired a defective copy of PRCA2 are particularly susceptible to such damages.
Humans who come into possession of a defective copy of the PRCA2 genes are prone to developing cancers. In this new trial, the aldehyde is shown to cause cell breakdown of BiRCA2 proteins. This effect lowers the level of proteins in human beings who come into possession of a defective copy of the Boehringer Ingelheim 2 (BRCA2) compound to below the level necessary for proper repair of genes and breaks off abnormal mechanism that could inhibit mutation and encourage cancers.
Approximately every 100th person can have a defective PRCA2 genetic defect and thus run the risks of having cancers of the breasts, ovaries, prostates and pancreas. "Professor Venkitaraman says, "Our research shows how chemical substances, to which we are becoming more and more vulnerable in everyday life, can raise the risks of illnesses such as cancers. "And it also explains why "the flaws in our stars" - the defective genetic material we were conceived with - may make some humans particularly vulnerable to the carcinogenic effect of these chemical substances.
Many Asians therefore produce a flush while drinking, but can also be particularly susceptible to the cancer-promoting effect.